Mathias V. Schmidt, PhD, Germany
Election statement:

The EBBS was the first scientific society I joined as a member and I have always found a scientific home there, as well as many friends. Especially during my time on the EBBS committee (2009 -2014) I was fascinated how important a society as the EBBS can be for shaping scientific policies as well as a platform of exchange of knowledge and ideas. The EBBS also plays an essential part in supporting young and advanced scientists to achieve their goals, attend meetings or organize symposia. After having organized the 2013EBBS meeting in Munich I realized how important it is to continue to keep this society strong and prospering, so that all the basic and clinical neuroscience researchers in Europe can connect. I would therefore be honored if I was given the chance to guide this society as President.

Short CV:
Education and Positions:

  • 1993-1996 Undergraduate and graduate training at the University of Bayreuth (Germany) and the University of Delaware (USA)
  • 2000-2003 PhD training in the Division of Medical Pharmacology LACDR, Leiden University (The Netherlands)
  • 2003-2004 Postdoctoral fellowship at the Max Planck Institute of Psychiatry, Munich, Germany
  • 2005-2010 Principle investigator at the research group “Molecular Stress Physiology” at the Max Planck Institute of Psychiatry, Munich, Germany
  • 2010- Research group leader at the Max Planck Institute of Psychiatry, “Neurobiology of Stress Resilience”, Munich, Germany

Research Area and Accomplishments:

Mathias Schmidt studies the impact of acute and chronic stress on the body during different developmental stages using a broad spectrum of approaches, ranging from different acute or chronic stress models in transgenic or knockout animals to pharmacological manipulations. By combining state-of-the-art behavioral, neuroendocrine and molecular readouts, he aims to develop novel pharmacological or genetic approaches to modulate, reverse or even prevent individual stress vulnerability. Dr. Schmidt has co-authored more than 100 articles and chapters with an H-index of 40, cited over 5000 times. His articles have been published in renowned journals as Nature Neuroscience, Nature Communications, Neuron, Molecular Psychiatry or PNAS. He received numerous honors and awards and is an active figure in the scientific community.

Selected publications of the last 5 years:

  1. Balsevich G, Häusl AS, Meyer CW, Karamihalev S, Feng X, Pöhlmann ML, Dournes C, Uribe-Marino A, Santarelli S, Labermaier C, Hafner K, Mao T, Breitsamer M, Theodoropoulou M, Namendorf C, Uhr M, Paez-Pereda M, Winter G, Hausch F, Chen A, Tschöp MH, Rein T, Gassen NC, Schmidt MV; Stress-responsive FKBP51 regulates AKT2-AS160 signaling and metabolic function; Nature Communications (2017), 8(1):1725
  2. Hartmann J, Dedic N, Pöhlmann ML, Häusl A, Karst H, Engelhardt C, Westerholz S, Wagner KV, Labermaier C, Hoeijmakers L, Kertokarijo M, Chen A, Joëls M, Deussing JM, Schmidt MV; Forebrain glutamatergic, but not GABAergic neurons mediate anxiogenic effects of the glucocorticoid receptor; Molecular Psychiatry (2017), 22(3):466-475
  3. Uribe-Marino A, Gassen NC, Wiesbeck MF, Balsevich G, Santarelli S, Solfrank B, Dournes C, Fries GR, Masana M, Labermaier C, Wang XD, Hafner K, Schmid B, Rein T, Chen A, Deussing JM, Schmidt MV; Prefrontal cortex corticotropin-releasing hormone receptor 1 conveys acute stress-induced executive dysfunction; Biological Psychiatry (2016), 80(10):743-753
  4. Gassen NC, Hartmann J, Zschocke J, Stepan J, Hafner K, Zellner A, Kirmeier T, Kollmannsberger L, Wagner KV, Dedic N, Balsevich G, Deussing JM, Kloiber S, Lucae S, Holsboer F, Eder M, Uhr M, Isig M, Schmidt MV*, Rein T*; Association of FKBP51 with priming autophagy pathways and mediating antidepressant treatment response: Evidence in cells, mice and humans; Plos Medicine (2014), 11(11):e1001755, *shared senior authorship
  5. Wang XD, Su YA, Wagner KV, Avrabos C, Scharf SH, Hartmann J, Wolf M, Liebl C, Kühne C, Wurst W, Holsboer F, Eder M, Deussing JM, Müller MB, Schmidt MV; Nectin-3 links CRHR1 signaling to stress-induced memory deficits and spine loss; Nature Neuroscience (2013), 16(6):706-13

Proposed by:

  • Barry Everitt, EBBS President 1998-2000 & President of FENS 2016-2018
  • Carmen Sandi, EBBS President 2011-12, President-elect of FENS